Mechanical effects of human cardiac troponin C mutation Gly159Asp in exchanged rabbit psoas fibres

Mechanical effects of human cardiac troponin C mutation Gly159Asp in exchanged rabbit psoas fibres

Sonraí bibleagrafaíochta
Príomhchruthaitheoirí:	Preston, L, Lipscomb, S, Robinson, P, Watkins, H, Redwood, C, Mogensen, J, Ashley, C
Formáid:	Conference item
Foilsithe / Cruthaithe:	2004

Míreanna comhchosúla

Functional effects of the DCM mutant Gly159Asp troponin C in skinned muscle fibres.
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DCM troponin C mutant Gly159Asp blunts the response to troponin phosphorylation.
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Mutations in fast skeletal troponin I, troponin T, and beta-tropomyosin that cause distal arthrogryposis all increase contractile function.
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A revised method of troponin exchange in permeabilised cardiac trabeculae using vanadate: functional consequences of a HCM-causing mutation in troponin I.
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Effects of hypertrophic cardiomyopathy mutations in troponin I on contractility of skinned cardiac muscle fibres
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Effects of troponin C isoform on the action of the cardiotonic agent EMD 57033.
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Effect of hypertrophic cardiomyopathy mutations in human cardiac muscle alpha -tropomyosin (Asp175Asn and Glu180Gly) on the regulatory properties of human cardiac troponin determined by in vitro motility assay.
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Dilated cardiomyopathy causing mutations in cardiac troponin T (TNT), cardiac troponin C (TNC), and alpha tropomyosin (TM) reduce CA(2+) sensitivity and maximum calcium activation
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Structure-function relationships of eight mutations in troponin and tropomyosin that cause dilated cardiomyopathy
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The effect of HCM mutations in troponin T and troponin I on the calcium binding of human cardiac troponin
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Using fluorescence emission spectroscopy to asses the Ca2+ affinity of reconstituted troponin and thin filaments containing cardiomyopathy causing mutations of troponin and alpha-tropomyosin
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CD14 C-159T and toll-like receptor 4 Asp299Gly polymorphisms in surviving meningococcal disease patients.
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The effect of the Asp175Asn and Glu180Gly TPM1 mutations on actin-myosin interaction during the ATPase cycle.
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Hypertrophic and dilated cardiomyopathycausing mutations of troponin and alpha-tropomyosin have opposite effects on thin filament calcium binding
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Investigating the Effect of Cardiomyopathy-Causing Mutations in Cardiac Troponin-T on Calcium Buffering In Situ
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Functional effects of mutations in troponin T and tropomyosin which cause dilated cardiomyopathy
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Altered inhibitory and troponin C-binding activities in cardiac troponin I mutants known to cause hypertrophic cardiomyopathy
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Mutations in fast skeletal troponin I, troponin T and beta-tropomyosin that cause distal arthrogryposis all enhance thin filament activation
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Dilated and hypertrophic cardiomyopathy mutations in troponin and alpha-tropomyosin have opposing effects on the calcium affinity of cardiac thin filaments.
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Severe disease expression of cardiac troponin C and T mutations in patients with idiopathic dilated cardiomyopathy.
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Comparison of the functional effects of mutations in troponin T which cause dilated and hypertrophic cardiomyopathies
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Two mutations in human cardiac troponin I known to cause hypertrophic cardiomyopathy have different effects on cardiac muscle contraction
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Investigation of troponin I mutations causing familial hypertrophic cardiomyopathy
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Bisphosphonate-functionalized cyclic Arg-Gly-Asp peptidomimetics
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Hypertrophic cardiomyopathy-causing Asp175asn and Glu180gly Tpm1 mutations shift tropomyosin strands further towards the open position during the ATPase cycle
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Hypertrophic cardiomyopathy-causing Asp175asn and Glu180gly Tpm1 mutations shift tropomyosin strands further towards the open position during the ATPase cycle.
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Tethering of Gly-Arg-Gly-Asp-Ser-Pro-Lys Peptides on Mg-Doped Hydroxyapatite
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Two mutations in troponin I that cause hypertrophic cardiomyopathy have contrasting effects on cardiac muscle contractility.
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Altered regulatory properties of human cardiac troponin I mutants that cause hypertrophic cardiomyopathy.
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Characterizations and molecular docking mechanism of the interactions between peptide FDGDF (Phe-Asp-Gly-Asp-Phe) and SOD enzyme
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A troponin I mutant known to cause hypertrophic cardiomyopathy shows reduced inhibition of Ca2+ independent tension
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Identification and functional analysis of cardiac troponin I as a novel disease gene in autosomal dominant familial dilated cardiomyopathy
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Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathy.
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Cardiac troponin I is a potential novel substrate for AMP-activated protein kinase
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An Asp7Gly substitution in PPARG is associated with decreased transcriptional activation activity.
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Effect of mutation Arg91Gly on the thermal stability of β-tropomyosin
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Histomorphometry of Ossification in Functionalised Ceramics with Tripeptide Arg-Gly-Asp (RGD): An In Vivo Study
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