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Mechanisms regulating myocardial redox state in chronic atrial fibrillation: the role of NADPH-oxidase and uncoupled nitric oxide synthases

Mechanisms regulating myocardial redox state in chronic atrial fibrillation: the role of NADPH-oxidase and uncoupled nitric oxide synthases

Bibliografiska uppgifter
Huvudupphovsmän: Margaritis, M, Antoniades, C, Demosthenous, M, Antonopoulos, A, Tousoulis, D, Bakogiannis, C, Lymperiadis, D, Reilly, S, Casadei, B, Stefanadis, C
Materialtyp: Conference item
Publicerad: 2010
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Liknande verk

  • Myocardial Redox State Predicts Post-Operative Clinical Outcome in Patients Undergoing Elective CABG: The Role of NADPH-Oxidase and Uncoupled Nitric Oxide Synthase
    av: Antoniades, C, et al.
    Publicerad: (2010)
  • Short-term preoperative treatment with atorvastatin improves myocardial redox state in patients undergoing elective CABG by affecting NADPH oxidase activity and NOS coupling
    av: Antoniades, C, et al.
    Publicerad: (2010)
  • Atorvastatin Treatment for Three Days Before CABG Improves Myocardial Redox State, by Modifying NADPH-Oxidase Activity and NOS Coupling
    av: Demosthenous, M, et al.
    Publicerad: (2010)
  • Preoperative atorvastatin treatment rapidly improves vein graft redox state by inhibiting rac1 and NADPH oxidase activity in patients undergoing elective CABG
    av: Antoniades, C, et al.
    Publicerad: (2010)
  • MYOCARDIAL O2-AND ONOO-GENERATION IN CHRONIC ATRIAL FIBRILLATION
    av: Margaritis, M, et al.
    Publicerad: (2011)

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