Amelioration of mitochondrial dysfunction-induced insulin resistance in differentiated 3T3-L1 adipocytes via inhibition of NF-kappa B pathways
A growing body of evidence suggests that activation of nuclear factor kappa B (NF-kappa B) signaling pathways is among the inflammatory mechanism involved in the development of insulin resistance and chronic low-grade inflammation in adipose tissues derived from obese animal and human subjects. Neve...
| Main Authors: | , , , , |
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| Format: | Article |
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MDPI AG
2014
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