GTPCH I overexpression decreases atherosclerotic lesion formation in apolipoprotein E-deficient/eNOS transgenic mice

مواد مشابهة

• Increased GTP-cyclohydrolase I expression but not vitamin C treatment restored accelerated atherosclerotic lesion formation in apolipoprotein E-deficient mice overexpressing endothelial nitric oxide synthase
  حسب: Takaya, T, وآخرون
  منشور في: (2004)
• Increased endothelial tetrahydrobiopterin levels in GTPCH-transgenic mice
  حسب: Alp, N, وآخرون
  منشور في: (2002)
• Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
  حسب: Bendall, J, وآخرون
  منشور في: (2005)
• Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
  حسب: Bendall, J, وآخرون
  منشور في: (2004)
• A specific role for eNOS-derived reactive oxygen species in atherosclerosis progression.
  حسب: Takaya, T, وآخرون
  منشور في: (2007)