Large-scale evidence that the cardiotoxicity of smoking is not significantly modified by the apolipoprotein E ε2/ε3/ε4 genotype

Large-scale evidence that the cardiotoxicity of smoking is not significantly modified by the apolipoprotein E ε2/ε3/ε4 genotype

Results from two small studies, involving a total of only 174 cases, have suggested that the increased risk of coronary heart disease conferred by cigarette smoking is substantially affected by genotype at the apolipoprotein E (APOE) ε2/ε3/ε4 polymorphism. We have established APOE genotypes in 4484...

पूर्ण विवरण

ग्रंथसूची विवरण
मुख्य लेखकों:	Keavney, B, Parish, S, Palmer, A, Clark, S, Youngman, L, Danesh, J, McKenzie, C, Delépine, M, Lathrop, M, Peto, R, Collins, R
स्वरूप:	Journal article
भाषा:	English
प्रकाशित:	2003

समान संसाधन

Large-scale evidence that the cardiotoxicity of smoking is not significantly modified by the apolipoprotein E epsilon2/epsilon3/epsilon4 genotype.
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प्रकाशित: (2003)

Meta-analysis for the Association of Apolipoprotein E ε2/ε3/ε4 Polymorphism with Coronary Heart Disease
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प्रकाशित: (2015-01-01)

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प्रकाशित: (2019-05-01)

ε2 allele and ε2-involved genotypes (ε2/ε2, ε2/ε3, and ε2/ε4) may confer the association of APOE genetic polymorphism with risks of nephropathy in type 2 diabetes: a meta-analysis
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Additive effect of LRP8/APOER2 R952Q variant to APOE ε2/ε3/ε4 genotype in modulating apolipoprotein E concentration and the risk of myocardial infarction: a case-control study
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Texture‐based morphometry in relation to apolipoprotein ε4 genotype, ageing and sex in a midlife population
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Salt free dyeing properties of cotton fabrics grafted with ε-polylysine(ε-PL)
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Fibrinogen and coronary heart disease: test of causality by 'Mendelian randomization'.
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