Inflammatory Ly-6C(hi) monocytes play an important role in the development of severe transplant arteriosclerosis in hyperlipidemic recipients.

ग्रंथसूची विवरण
मुख्य लेखकों:	Schiopu, A, Nadig, SN, Cotoi, O, Hester, J, van Rooijen, N, Wood, K
स्वरूप:	Journal article
भाषा:	English
प्रकाशित:	2012

Inflammatory Ly-6Chi monocytes play an important role in the development of severe transplant arteriosclerosis in hyperlipidemic recipients
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Ly-6C(hi) monocytes: a potential target for preventing transplant arteriosclerosis?
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Low-dose rapamycin treatment increases the ability of human regulatory T cells to inhibit transplant arteriosclerosis in vivo.
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In vivo prevention of transplant arteriosclerosis by ex vivo-expanded human regulatory T cells.
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Indirect allorecognition can play an important role in the development of transplant arteriosclerosis.
द्वारा: Ensminger, S, और अन्य
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Regulation of transplant arteriosclerosis by CD25+CD4+ T cells generated to alloantigen in vivo.
द्वारा: Warnecke, G, और अन्य
प्रकाशित: (2007)

Specific depletion of Ly6C(hi) inflammatory monocytes prevents immunopathology in experimental cerebral malaria.
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Indirect allorecognition plays a pivotal role in the pathogenesis of transplant arteriosclerosis.
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Complex arterial reconstruction for pancreas transplantation in recipients with advanced arteriosclerosis.
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CD4+ regulatory T cells generated in vitro with IFN-{gamma} and allogeneic APC inhibit transplant arteriosclerosis.
द्वारा: Warnecke, G, और अन्य
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Ex vivo expanded human treg inhibit the development of transplant arteriosclerosis in a humanized in vivo model
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Mouse endothelial CD40 expression does not play a role during the development of transplant arteriosclerosis.
द्वारा: Ensminger, S, और अन्य
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The persistence of transplant arteriosclerosis despite CD154 blockade.
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Increased transplant arteriosclerosis in the absence of CCR7 is associated with reduced expression of Foxp3.
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Ly6C hi Monocytes Are Metabolically Reprogrammed in the Blood during Inflammatory Stimulation and Require Intact OxPhos for Chemotaxis and Monocyte to Macrophage Differentiation
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The Properties of Proinflammatory Ly6C<sup>hi</sup> Monocytes Are Differentially Shaped by Parasitic and Bacterial Liver Infections
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Type I interferon signaling regulates Ly6C(hi) monocytes and neutrophils during acute viral pneumonia in mice.
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Ly6C<sup>hi</sup> Monocytes Are Metabolically Reprogrammed in the Blood during Inflammatory Stimulation and Require Intact OxPhos for Chemotaxis and Monocyte to Macrophage Differentiation
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Immune-Mediated Vascular Injury and Dysfunction in Transplant Arteriosclerosis
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Comparison of the effects of exposure to a single or multiple donor alloantigens in the development of transplant arteriosclerosis.
द्वारा: Ensminger, S, और अन्य
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CD8+ T cells contribute to the development of transplant arteriosclerosis despite CD154 blockade.
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Eosinophils are major effector cells for the development of transplant arteriosclerosis and depend on the presence of IL-4.
द्वारा: Ensminger, S, और अन्य
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Ly6C- Monocytes Regulate Parasite-Induced Liver Inflammation by Inducing the Differentiation of Pathogenic Ly6C+ Monocytes into Macrophages.
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Kinetics of transplant arteriosclerosis in MHC-Class I mismatched and fully allogeneic mouse aortic allografts.
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Tadalafil attenuates graft arteriosclerosis of aortic transplant in a rat model
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Transplantation tolerance: lessons from experimental rodent models.
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MSCs Contribute to the Conversion of Ly6Chigh Monocytes into Ly6Clow Subsets under AMI
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Dampened inflammatory signalling and myeloid-derived suppressor-like cell accumulation reduces circulating monocytic HLA-DR density and associates with malignancy risk in long-term renal transplant recipients
द्वारा: Bottomley, M, और अन्य
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NFAT1 Regulates Ly6Chi Monocyte Recruitment to the CNS and Plays an Essential Role in Resistance to Toxoplasma gondii Infection
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Dampened inflammatory signalling and myeloid-derived suppressor-like cell accumulation reduces circulating monocytic HLA-DR density and may associate with malignancy risk in long-term renal transplant recipients
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Critical role for IL-4 in the development of transplant arteriosclerosis in the absence of CD40-CD154 costimulation.
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C-Reactive Protein and Arteriosclerosis
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HI in the protocluster environment at z > 2: absorbing haloes and the Ly alpha forest
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Triggering of NOD2 Receptor Converts Inflammatory Ly6Chigh into Ly6Clow Monocytes with Patrolling Properties
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प्रकाशित: (2017-08-01)

Inflammatory Ly-6C(hi) monocytes play an important role in the development of severe transplant arteriosclerosis in hyperlipidemic recipients.

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