Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels

Detalhes bibliográficos
Principais autores:	Antoniades, C, Shirodaria, C, Leeson, P, Tousoulis, D, Pillai, R, Ratnatunga, C, Vasiliadou, C, Antonopoulos, A, Stefanadis, C, Channon, K
Formato:	Journal article
Publicado em:	2007

Plasma asymmetrical-dimethyl-arginine (ADMA) as a regulator of vascular redox and nitric oxide bioavailability in human vessels
por: Antoniades, C, et al.
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Plasma asymmetrical-dimethyl-arginine affects eNOS vascular superoxide radical production and nitric oxide bioavailability in human arteries and veins
por: Antoniades, C, et al.
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Association of plasma asymmetrical dimethylarginine (ADMA) with elevated vascular superoxide production and endothelial nitric oxide synthase uncoupling: implications for endothelial function in human atherosclerosis.
por: Antoniades, C, et al.
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Genetic Polymorphism On Methyl-Tetrahydrofolate-Reductase Affects Vascular Superoxide Generation and Nitric Oxide Bioavailability By Regulating eNOS Coupling in Human Arteries and Veins
por: Antoniades, C, et al.
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Genetic variability of MTHFR affects vascular redox and nitric oxide bioavailability in human vessels, by modifying eNOS coupling
por: Antoniades, C, et al.
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The common genetic polymorphism C677T of methyltetrahydrofolate reductase regulates nitric oxide bioavailability and vascular superoxide production via modulation of eNOS coupling in human vessels
por: Antoniades, C, et al.
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5-methyl-tetrahydrofolic acid increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling in human vessels in vivo: Effects on intracellular tetrahydrobiopterin oxidation
por: Antoniades, C, et al.
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5-methyl-tetrahydrofolate increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling and preventing the intracellular oxidation of tetrahy-drobiopterin, in human vessels in vivo
por: Antoniades, C, et al.
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Chronic treatment with folic acid improves nitric oxide bioavailability and decreases superoxide production in human vessels, through tetrahydrobiopterin-mediated improvement of eNOS coupling
por: Antoniades, C, et al.
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Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling
por: Antoniades, C, et al.
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Soluble CD40-ligand levels are associated with vascular superoxide production and eNOS coupling in human vessels: Interrelationships with homocysteine biosynthetic pathway
por: Antoniades, C, et al.
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5-Methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production by improving eNOS coupling and increasing tetrahydrobiopterin bioavailability in human vessels in vivo
por: Antoniades, C, et al.
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Genetic Variability of Methyl-Tetrahydrofolate-Reductase Modifies eNOS Coupling in Human Arteries and Veins: Effects on Vascular Superoxide Generation and Nitric Oxide Bioavailabllity
por: Antoniades, C, et al.
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5-methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production in vessels from patients with coronary artery disease
por: Antoniades, C, et al.
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5-methyltetrahydrofolate rapidly improves endothelial function and decreases superoxide production in human vessels: effects on vascular tetrahydrobiopterin availability and endothelial nitric oxide synthase coupling.
por: Antoniades, C, et al.
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Atorvastatin rapidly modifies vascular redox state and improves nitric oxide bioavailability in human vessels obtained from patients with advanced atherosclerosis
por: Demosthenous, M, et al.
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Direct improvement of endothelial function and superoxide production in human vessels, by oral folic acid treatment: A double blind placebo controlled study
por: Antoniades, C, et al.
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Relationships between vascular/plasma biopterins and endothelial nitric oxide synthase coupling, endothelial function and inflammation, in human atherosclerosis
por: Antoniades, C, et al.
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Intravenous infusion of 5-methyl-tetrahydrofolate rapidly improves eNOS Coupling. In human vessels by increasing tetrahydrobiopterin bioavailability
por: Antoniades, C, et al.
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Low- and high- dose treatment with folic acid, equally improve endothelial function and decrease superoxide production in human vessels via tetrahydrobiopterin-mediated eNOS coupling
por: Antoniades, C, et al.
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Serum adiponectin regulates vascular O2-generation and NO bioavailability in patients with coronary atherosclerosis
por: Antoniades, C, et al.
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Chronic treatment with folic acid improves endothelial function and decreases superoxide production in human vessels. A double blind placebo control study
por: Shirodaria, C, et al.
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Associations Between Serum Adiponectin, Vascular O2-Generation and NO Bioavailability In Patients With Coronary Atherosclerosis
por: Antoniades, C, et al.
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Superoxide production by NAD(P)H oxidase in human blood vessels: Functional interaction with nitric oxide
por: Guzik, T, et al.
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Direct effects of 5-methyl-tetrahydrofolate on vascular endotheliurn and superoxide production in human arteries and veins
por: Antoniades, C, et al.
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Altered plasma vs. vascular biopterins in human atherosclerosis reveal relationships between endothelial nitric oxide synthase coupling, endothelial function and inflammation
por: Antomades, C, et al.
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Plasma levels of sCD40-ligand interact with homocysteine biosynthetic pathway and predict eNOS coupling in human vessels: effects on vascular redox state
por: Antoniades, C, et al.
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RAPID, DIRECT EFFECTS OF STATIN TREATMENT ON ARTERIAL REDOX STATE AND NITRIC OXIDE BIOAVAILABILITY IN HUMAN ATHEROSCLEROSIS VIA TETRAHYDROBIOPTERIN-MEDIATED ENOS COUPLING
por: Antoniades, C, et al.
Publicado em: (2011)

Effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
por: Antoniades, C, et al.
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Is vascular superoxide triggering adiponectin biosynthesis in perivascular adipose tissue in patients with atherosclerosis?
por: Bakogiannis, C, et al.
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Altered plasma versus vascular biopterins in human atherosclerosis reveal relationships between endothelial nitric oxide synthase coupling, endothelial function, and inflammation.
por: Antoniades, C, et al.
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Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin-mediated endothelial nitric oxide synthase coupling.
por: Antoniades, C, et al.
Publicado em: (2011)

Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin- mediated endothelial nitric oxide synthase coupling
por: Antoniades, C, et al.
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Superoxide generation by NAD(P)H oxidase in human sphenous vein: Interaction with nitric oxide
por: Guzik, T, et al.
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Rapid effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
por: Antoniades, C, et al.
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Nitric oxide modulates superoxide release and peroxynitrite formation in human blood vessels.
por: Guzik, T, et al.
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Pre-operative flow mediated dilatation in patients undergoing coronary artery bypass surgery: Prediction of NO-bioavailability and vascular superoxide production in arterial and vein grafts
por: Antoniades, C, et al.
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Preoperative evaluation of flow-mediated dilatation in the left radial artery or the right brachial artery, as a predictor of nitric-oxide bioavailability in coronary artery bypass grafts
por: Antoniades, C, et al.
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Oral Atorvastatin Treatment Improves NO Bioavailability and Vascular Redox State in the Human Arterial Wall, via a Tetrahydrobiopterin-Mediated Improvement of eNOS Coupling
por: Antoniades, C, et al.
Publicado em: (2011)

Irbesartan lowers superoxide levels and increases nitric oxide bioavailability in blood vessels from spontaneously hypertensive stroke-prone rats.
por: Brosnan, M, et al.
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Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels

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