Histone H2A monoubiquitylation and p38-MAPKs regulate immediate-early gene-like reactivation of latent retrovirus HTLV-1
It is not understood how the human T cell leukemia virus human T-lymphotropic virus-1 (HTLV-1), a retrovirus, regulates the in vivo balance between transcriptional latency and reactivation. The HTLV-1 proviral plus-strand is typically transcriptionally silent in freshly isolated peripheral blood mon...
Príomhchruthaitheoirí: | Kulkarni, A, Taylor, G, Klose, R, Schofield, C, Bangham, C |
---|---|
Formáid: | Journal article |
Teanga: | English |
Foilsithe / Cruthaithe: |
American Society for Clinical Investigation
2018
|
Míreanna comhchosúla
Míreanna comhchosúla
-
Glucose metabolism and oxygen availability govern reactivation from latency of the human retrovirus HTLV-1
de réir: Kulkarni, A, et al.
Foilsithe / Cruthaithe: (2017) -
Histone deacetylase inhibitors increase virus gene expression but decrease CD8+ cell antiviral function in HTLV-1 infection.
de réir: Mosley, A, et al.
Foilsithe / Cruthaithe: (2006) -
Clinical, histological and immunological features of HTLV-1-associated infective dermatitis
de réir: Hlela, C, et al.
Foilsithe / Cruthaithe: (2010) -
Clonality of HTLV-2 in natural infection
de réir: Melamed, A, et al.
Foilsithe / Cruthaithe: (2014) -
The p38 MAPK Pathway in Rheumatoid Arthritis: A Sideways Look.
de réir: Clark, A, et al.
Foilsithe / Cruthaithe: (2012)